GC182 Chopped And Stabbed Wound In Gang Fight Nerves And Vascular Injury; Classification Of Injuries
Chopped and stabbed wounds sustained in gang fights involving nerve and vascular damage are classified based on wound mechanism (incised, stab, or chop), depth of penetration, and severity of neurovascular injury to guide medicolegal documentation and surgical management.
Chopped and Stabbed Wound in Gang Fight: Nerves and Vascular Injury; Classification of Injuries
Lecture Map
This lecture teaches you to manage a patient who arrives in the emergency department after a gang fight with chopping and stabbing injuries. The core philosophy is: treat what kills first, then save the limb, then reconstruct function. The lecture integrates ATLS principles (primary/secondary survey), biomechanics of injury (blunt vs penetrating), wound classification (chopped vs stabbed), and the specific management of peripheral vascular and nerve injuries in the extremities. Understanding why a partially severed artery bleeds more than a completely severed one, and why Seddon's classification of nerve injury determines prognosis, are the two conceptual pillars examiners love to test.
- Understand the correct priority in the management of patients with multiple injuries
- Recognize the differences between chopped (cut) and stabbed wound (penetrating injury versus blunt injury)
- Understand the basic principles of evaluation of these injuries
- Basic principles on blood vessel and peripheral nerve injuries
Core Concepts and Mechanisms
"ATLS program is to treat the greatest threat to life first" [1]
"The lack of a definitive diagnosis and a detailed history should not slow the application of indicated treatment for life-threatening injury, with the most time-critical interventions performed early" [1]
Why this matters from first principles: Trauma patients often present unconscious, with incomplete history, and multiple simultaneous injuries. Without a systematic approach, life-threatening injuries get missed because clinicians focus on the most dramatic-looking wound (e.g. a chopped limb) rather than the most dangerous one (e.g. a tension pneumothorax). ATLS gives you a reproducible algorithm so you never skip a lethal condition.
The continuous process: ATLS starts at the point of injury and continues through:
- Initial assessment
- Life-saving intervention
- Re-evaluation
- Stabilisation
- Transfer to specialised care (trauma centre) [1]
The primary survey identifies and treats immediately life-threatening conditions. It runs simultaneously with resuscitation.
| Step | Focus | Key Actions | Why |
|---|---|---|---|
| A | Airway + C-spine protection | Check for patent airway, stridor; apply cervical collar | Airway obstruction kills in minutes. C-spine must be protected because neck manipulation in an unstable fracture → quadriplegia |
| B | Breathing + Ventilation | Auscultate for equal breath sounds, look for flail segment | Tension pneumothorax, open pneumothorax, massive haemothorax can cause rapid death if not treated |
| C | Circulation + Haemorrhage control | BP/Pulse, IV access, warmed fluids, stop external bleeding | Haemorrhagic shock is the commonest preventable cause of trauma death |
| D | Disability | GCS, pupil responses | Expanding intracranial haematoma needs urgent intervention |
| E | Exposure + Environmental control | Undress patient, prevent hypothermia | Missed injuries under clothing; hypothermia worsens coagulopathy (trauma triad of death: hypothermia, acidosis, coagulopathy) |
Airway Mis-management Pitfalls
Common causes of airway mismanagement in trauma (from lecture slides): [1]
- Failure to recognise the inadequate airway
- Failure to establish a clear airway with/without airway device
- Failure to recognise incorrect placement of airway device
- Displacement of a previously established airway
- Failure to recognise the need for ventilation
- Aspiration of gastric contents
These are actual exam pitfalls — examiners test whether you can identify the specific airway failure mode.
"Should occur simultaneously with primary survey" [1]
- Oxygenation and ventilation
- Shock management: intravenous lines, warmed IV fluids
- Management of life-threatening problems identified in primary survey [1]
Why warmed fluids? Cold fluids worsen hypothermia → impair clotting cascade → more bleeding → more transfusion needed (vicious cycle).
| Adjunct | Purpose |
|---|---|
| CXR, Pelvis XR, C-spine XRs | Detect pneumothorax, haemothorax, pelvic fracture, cervical spine fracture |
| FAST scan / DPL | Detect intra-abdominal free fluid (blood) — guides decision for laparotomy |
| NG tube | Decompress stomach, detect upper GI bleeding |
| Urine output monitoring | Best real-time indicator of organ perfusion |
| Monitors: BP/P/RR, oximeter, ECG, GCS | Continuous reassessment |
Visual recognition (adjuncts slide): the photograph captioned "Intubated and chest drain" shows simultaneous airway control and pleural decompression/drainage — adjuncts are not paperwork after ABC; they run with resuscitation. [1]
FAST Scan
FAST = Focused Assessment with Sonography for Trauma. It checks 4 areas: pericardial window, Morrison's pouch (hepatorenal), splenorenal space, and pelvis (pouch of Douglas/rectovesical). Positive FAST in an unstable patient → laparotomy. [2]
"Begins when: Primary survey ABCDE is complete and secured; Resuscitation efforts are well established; Patient's vital signs are stabilised" [1]
What it involves:
- Head-to-toe, front-to-back examination
- Log roll with neck collar on — to examine the back and spine
- Spinal injury may result in motor/sensory loss, incontinence, lax anal tone
- Spinal shock = flaccid paralysis, autonomic dysfunction [1]
- Systematic examination: Head, neck, chest, abdomen, pelvis, perineum, extremities [1]
AMPLE History:
| Letter | Stands For |
|---|---|
| A | Allergies |
| M | Medications currently used |
| P | Past illness / Pregnancy |
| L | Last meal |
| E | Events / Environment leading to injuries |
Why "Last meal"? A full stomach increases aspiration risk during intubation/anaesthesia. Knowing the last meal time helps the anaesthetist decide on rapid sequence induction.
Biomechanics of Injury [1]
| Type | Examples | Mechanism |
|---|---|---|
| Blunt trauma | Road traffic accidents | Force dissipated over wide area |
| Penetrating trauma | Knife, gunshot | Kinetic energy transferred along weapon track |
| Burns | Scalds, chemicals | Thermal/chemical tissue destruction |
| Blast injuries | Bombs | Combined pressure wave + projectile + thermal |
"The force is dissipated over a wide area, minimising energy transfer at one spot"
| Force | Direction | Effect | Classic Example |
|---|---|---|---|
| Shearing | Two forces in opposite directions | Tears organs at tethered points (acceleration-deceleration) | Fall from height → duodenojejunal junction, spleen, liver tear |
| Tension | Force < 90° to surface | Avulsions, flap formation → tissue necrosis | Degloving injury |
| Compression | Force at 90° to surface | Contusion/haematoma; raised internal pressure → rupture of hollow viscus | Seat-belt compression of bowel |
This is a classic exam list — learn it systematically from head to toe:
Head injury → Cervical injury → Vertebral wedge fracture → Pelvic fracture → Tracheo-bronchial dislocation → Pneumothorax/haemothorax → Rupture or dissection of aorta → Liver or spleen laceration → Rupture of bowel → Lower limb/ankle/fracture of calcaneum or metatarsals
Why calcaneum? In axial loading (landing feet-first), force transmits directly through the heel. Calcaneal fractures are pathognomonic of falls from height and should prompt you to check the lumbar spine for associated compression fractures (same axial loading mechanism).
"Kinetic Energy (KE = mass/2 × velocity²) is transferred to tissues surrounding the track of the weapon or missile"
Why velocity matters more than mass: KE depends on velocity squared. A bullet (small mass, very high velocity) transfers far more energy than a knife (larger presenting area, low velocity). This is why gunshot wounds cause massive cavitation while knife wounds cause a narrow track.
Factors determining injury severity:
- Mean presenting area of weapon
- Tendency of weapon to deform, change pathway, or fragment
- Density/characteristics of tissue (solid organs like liver sustain more damage than lungs)
- Impact velocity/damage to neighbouring tissues
Three consequences of penetrating trauma: [1]
- Functional and mechanical disruption of neighbouring tissues and energy transfer
- A core of covering clothing is carried deep into the wound → contamination
- In gunshot wounds: exit wound is usually larger than entry wound (temporary cavitation effect extends along wound track; some missiles fragment on contact)
High Yield Definition
Cut wounds (incised/chopped wounds) = wounds caused by a sharp-edged weapon/instrument
Stab wounds = wounds where the depth of the wound is longer than the width of the wound
Both are classified as penetrating/sharp injury (NOT blunt injury)
May be difficult to differentiate — depends on mode of injury [1]
| Feature | Chopped (Incised/Cut) Wound | Stab Wound |
|---|---|---|
| Depth vs Width | Width ≥ Depth | Depth > Width |
| Weapon type | Sharp-edged (cleaver, machete) | Pointed (knife, screwdriver) |
| Injury pattern | Wide tissue exposure, may be superficial | Narrow entry, deep penetration |
| Risk | Bleeding, tendon/nerve transection | Deep organ injury, may seal externally |
| Classification | Penetrating/sharp | Penetrating/sharp |
Documentation of Wounds [1]
"It is important to classify injuries correctly, to note the details e.g. location, size, shape, direction, presence of foreign bodies etc."
"The wound should be photographed or sketched before surgical repair" [1]
Why documentation matters: In gang fight cases, these become medico-legal evidence. Accurate documentation can determine whether it was self-inflicted or assault, and helps forensic pathologists reconstruct the event. Past paper 2020 SAQ Q3 directly tests this [7].
Key features to document for a stab wound:
- Location (anatomical site, referenced to landmarks)
- Size (length × width of skin wound)
- Shape (elliptical, slit-like, irregular)
- Direction/depth (angle of entry if assessable)
- Wound edges (clean/ragged — clean = sharp instrument)
- Presence of foreign bodies
- Surrounding bruising or abrasion ("collar" around stab wound = blunt component)
Regional Penetrating Injuries
Life-threatening conditions need to be ruled out, presume cardiac and lung injuries until proven otherwise:
- Cardiac tamponade
- Open pneumothorax
- Massive haemothorax
Why presume until proven otherwise? A small external wound can mask a lethal internal injury. A stab wound in the "cardiac box" (bordered by clavicles, nipple lines, and costal margin) has a high probability of cardiac injury.
Key point from slides: "Big wound but less chance of injury to the underlying organs (not true)" [1] — The lecture explicitly debunks the myth. A large chopping wound of the chest wall may look dramatic but the blade may not have penetrated deeply. Conversely, a tiny stab wound may reach the heart.
Management of pneumothorax/haemothorax: [1]
- Give Oxygen
- Insert chest tube
- Treat local wound
- Urgent thoracotomy if massive bleeding
What the chest trauma photograph shows: a penetrating/open lateral chest wound with a chest tube already in situ and blood visible in the tubing — the slide's four management points are not theoretical; they are the sequence you should already be executing when this image appears in resus. [1]
When is thoracotomy indicated? If chest tube drainage shows > 1500 mL immediately or > 200 mL/hour for 2–4 hours (massive haemothorax). Also for cardiac tamponade unresponsive to pericardiocentesis.
Exsanguinating external bleeding (may be from external jugular vein)
Expanding haematoma → endotracheal intubation to protect airway → operative exploration
If unsure of diagnosis: wound deeper than platysma → requires exploration
Why platysma is the key layer: The platysma is a thin superficial muscle in the neck. If a wound penetrates deeper than platysma, it can potentially injure major vessels (carotid, jugular), the airway (trachea, larynx), the oesophagus, or nerves (vagus, recurrent laryngeal). Wounds superficial to platysma can be managed conservatively.
Surface anatomy matters:
- Thoracoabdominal area (nipple line to costal margin) — remember the diaphragm! A stab wound here may injure both chest and abdominal organs
- Anterior abdomen
- Flank
- Back
Management algorithm:
| Haemodynamic Status | Management |
|---|---|
| Stable | CT → Local wound exploration in OR → Laparoscopy → Laparotomy |
| Unstable | Immediate laparotomy |
Why CT first if stable? CT triple-contrast can identify specific organ injuries, guide selective non-operative management, and avoid unnecessary laparotomy. But an unstable patient cannot wait — they need surgical haemostasis immediately.
Visual recognition: one slide shows a large gaping anterior abdominal laceration (transverse/oblique with active bleeding) — dramatic surface findings still do not dictate the algorithm; haemodynamic stability does. Another operative photograph is captioned exploratory laparotomy for haemostasis and repair of caecum, with bowel loops exposed under retractors — the teaching point is source control + hollow-viscus repair, not a leisurely inventory of every bruise. [1]
Priority Principle
"Identify and treat life-threatening injuries before limb salvage" [1]
This is a core exam principle. You must stabilize the patient (ABCDE) before focusing on the mangled limb. A dead patient with a perfectly repaired arm is still a dead patient.
Structures to Assess (Systematic Approach) [1]
Skin → Arteries → Veins → Nerves → Muscle & Tendons → Bones and Joints
This is the order you should document. Each structure requires specific examination:
| Structure | How to Assess | Why |
|---|---|---|
| Skin | Wound size, contamination, viability of edges | Determines closure method |
| Arteries | Distal pulses, capillary refill, Doppler, ± angiogram | Ischaemia → tissue death if not repaired within 6h |
| Veins | Bleeding pattern (dark, steady ooze vs bright pulsatile) | Venous bleeding usually controlled by pressure |
| Nerves | Motor and sensory exam distal to wound | Determines if nerve repair needed |
| Muscle/Tendons | Active movement against resistance | Tendon injuries missed if not actively tested |
| Bones/Joints | Deformity, crepitus, XR | Open fracture management needed |
Vascular Injury
This is a classic exam concept:
Partially severed artery → incomplete contraction/retraction → massive bleeding & formation of pseudoaneurysm
Completely severed (divided) artery → contraction and retraction → less bleeding
Why does this paradox occur? An artery has smooth muscle in its wall (tunica media). When completely transected, the circular smooth muscle can contract fully, and the elastic recoil causes the cut ends to retract into surrounding tissue, partially sealing the vessel. When only partially cut, the remaining intact wall prevents full contraction and retraction — the wound in the vessel wall is held open by the intact portion, causing more profuse bleeding. The partially cut vessel can also develop a pseudoaneurysm (contained rupture) because blood continues to pulse through the defect.
High Yield Exam Point
A partially severed artery bleeds MORE than a completely severed artery because incomplete transection prevents full muscular contraction and retraction of the vessel. This is a classic viva/written question discriminator. [1]
- Right axillary penetrating injury — can damage axillary artery and brachial plexus [1]
- Transected brachial artery & nerve injuries — requires interposition vein graft [1]
- RTA with fracture and free-floating knee & pulseless leg → External fixation + angiogram showing abrupt cut-off at distal SFA → SFA to below-knee popliteal artery reversed long-saphenous vein bypass graft [1]
What the operative / imaging sequence shows: [1]
- Axilla: wide open right axillary wound with deep bleeding — treat as combined vascular + brachial-plexus territory until proven otherwise; examine the whole limb, not only the hole.
- Arm: green arrows on a transected brachial artery with adjacent nerve ends in the same field — proximal/distal vascular control and planned nerve repair travel together.
- Floating knee + pulseless leg: long-leg radiographs with comminuted distal femur / proximal tibia–fibula disruption → spanning external fixator first for skeletal stability in a pulseless limb.
- Angiogram: abrupt cutoff at distal SFA (arrowed) → operative exploration of SFA and below-knee popliteal with vessel loops — anatomy of the bypass targets, not just “do an angiogram.”
- Two weeks later: 3D CT still shows the severe peri-knee comminution while the limb remains in the fixator frame — temporary spanning fixation buys time for soft-tissue and vascular recovery before definitive reconstruction.
| Technique | When Used |
|---|---|
| Primary repair | Clean transection with minimal gap; proximal and distal control of arteries first |
| Interposition vein graft | Gap too large for primary repair; use reversed saphenous vein |
| Bypass graft | Vessel too damaged or extensive; SFA to below-knee popliteal reversed LSV graft |
Why "proximal and distal control first"? Before you open a vascular injury, you must have clamps or tourniquets on both sides to prevent catastrophic haemorrhage during repair.
- Save life
- Haemostasis and blood replacement
- Primary repair (proximal and distal control of arteries first)
- Limb salvage
- Rehabilitation
- Late reconstruction
Peripheral Nerve Injury
"A peripheral nerve is an enclosed bundle of axons. A neuron is an electrically excitable cell that processes and transmits information through electrical and chemical signals."
"A nerve provides a common pathway for electrochemical impulses transmitted along each axon to peripheral organs." [1]
A peripheral nerve has three connective tissue layers (outermost → innermost):
- Epineurium — outermost, surrounds entire nerve
- Perineurium — surrounds fascicles (bundles of axons)
- Endoneurium — surrounds individual axons
Blood vessels are also present within the nerve between and around the fascicles, supplying the vasa nervorum. [1]
Understanding these layers is critical because the classification of nerve injury is based on which layers are disrupted.
This is the most commonly examined nerve injury classification:
| Grade | Name | Pathology | Connective Tissue | Recovery | Treatment |
|---|---|---|---|---|---|
| Class I | Neuropraxia | Minor blunt injury → temporary conduction block; axonal system intact | All intact | Complete functional recovery expected (days to weeks) | Observation |
| Class II | Axonotmesis | Severe trauma → interruption of axonal system; distal axon dies, myelin sheath disintegrates | Endoneurium intact | Eventual good functional outcome; takes months | Observation (axons regrow along intact endoneurial tubes) |
| Class III | Neurotmesis | Total disruption, laceration, or extreme traction of nerve fibre | All layers disrupted | Distal Wallerian disintegration and axonal death; poor functional outcome | Surgical repair indicated |
Key Concept: Wallerian Degeneration
When an axon is cut, the portion distal to the injury undergoes Wallerian degeneration: the axon and myelin sheath break down and are cleared by macrophages. The proximal stump then sends out axon sprouts that attempt to regrow. In axonotmesis, the intact endoneurial tubes act as "highways" guiding the sprouts to the correct target — hence good recovery. In neurotmesis, there are no tubes left, so sprouts grow randomly, forming a painful neuroma with poor functional recovery — hence surgical repair (epineurial or fascicular repair) is needed.
"Axons sprouts to repair at 1mm per day" [1]
Clinical implication: If a nerve is injured at a point 30 cm from the target muscle, recovery takes approximately 300 days (~10 months). This is why proximal nerve injuries have worse prognosis — the axon must grow a longer distance, and the target muscle may undergo irreversible atrophy before reinnervation occurs (> 12–18 months without reinnervation → permanent muscle atrophy).
While the lecture focuses on Seddon, examiners may reference Sunderland's 5-grade system. Here's how they map:
| Seddon | Sunderland | Structure Damaged |
|---|---|---|
| Neuropraxia | Grade I | Myelin only (focal demyelination) |
| Axonotmesis | Grade II | Axon + myelin (endoneurium intact) |
| Neurotmesis | Grade III | Axon + myelin + endoneurium |
| Neurotmesis | Grade IV | + perineurium |
| Neurotmesis | Grade V | Complete transection (all layers) |
Clinical Approach Summary
- Mechanism: Chopping weapon (cleaver, machete) vs stabbing weapon (knife, screwdriver) vs gunshot
- Time since injury: Critical for ischaemia time (golden 6 hours for vascular repair)
- Symptoms: Pain, numbness, weakness, inability to move fingers/toes
- AMPLE history for ATLS
- Primary survey ABCDE — always first
- Secondary survey — systematic head-to-toe
- Limb examination:
- Skin: wound characteristics, contamination
- Vascular: 5 P's of ischaemia — Pain, Pallor, Pulselessness, Paraesthesia, Paralysis (and Poikilothermia = cold limb)
- Nerve: test motor and sensory function of each major nerve
- Tendon: test active movement
- Bone: deformity, XR
| Investigation | Purpose |
|---|---|
| Trauma XR series (CXR, Pelvis, C-spine) | Screen for life-threatening injuries |
| FAST scan | Detect intra-abdominal free fluid |
| CT (if stable) | Detailed assessment of chest/abdomen/head |
| Angiography (CT angiogram or conventional) | Confirm and localize vascular injury |
| Doppler ultrasound | Bedside assessment of arterial flow |
| Nerve conduction studies / EMG | Not acutely — used 3–4 weeks later to assess nerve injury grade |
Immediate:
- ATLS primary survey and resuscitation
- Direct pressure for haemorrhage control (tourniquet as last resort in extremity)
- Blood replacement (cross-match, activate massive transfusion protocol if needed)
Definitive:
- Chest: chest tube → thoracotomy if massive haemothorax
- Abdomen: laparotomy if unstable
- Limb vascular: proximal/distal control → primary repair or vein graft
- Nerve: primary repair if neurotmesis (clean sharp transection), or delayed repair (2–3 weeks) if wound contaminated
- Open fracture: debridement, external fixation, antibiotics (Gustilo-Anderson classification) [3]
- Tetanus prophylaxis
Rehabilitation:
- Physiotherapy for joint mobilisation
- Occupational therapy for hand function
- Serial nerve examination to monitor recovery (Tinel's sign advancing distally = good sign)
The 2020 SAQ Q3 specifically tests this. Key differentiators:
| Feature | Self-Inflicted | Assault |
|---|---|---|
| Location | Accessible areas (wrist, forearm) | Any area including back, non-dominant side |
| Number | Multiple superficial "hesitation marks" | Usually fewer, deeper wounds |
| Depth | Superficial, tentative | Deep, forceful |
| Direction | Parallel, regular | Irregular, varied angles |
| Defence wounds | Absent | Present (palmar surface of hands, forearms) |
| Clothing | Often pulled away from wound site | Clothing may be cut through |
Exam Intelligence
-
"Blunt vs penetrating": Both chopped AND stabbed wounds are penetrating/sharp injuries — examiners love to trick you into calling a chopped wound "blunt" because a cleaver is heavy.
-
Partially vs completely severed artery: Remember the paradox — partial = MORE bleeding. This is tested in MCQs as a "which bleeds more" question.
-
Primary survey before limb: Never describe limb repair before confirming ABCDE is complete. In SAQs, always state "after stabilization" before discussing definitive limb management.
-
Neuropraxia vs neurotmesis: If the question says "complete functional recovery expected," it's neuropraxia. If "surgical repair indicated," it's neurotmesis. Axonotmesis is the middle ground — good recovery but takes months.
-
Axon growth rate: 1 mm/day — this comes up in calculations of expected recovery time.
-
Stab wound of chest: Always rule out cardiac tamponade, open pneumothorax, massive haemothorax — even if the wound looks minor.
-
Wound deeper than platysma in the neck: This is the threshold for mandatory exploration.
-
Exit wound > entry wound in gunshot: Due to cavitation effect.
-
FAST scan: The lecture mentions both FAST and DPL as adjuncts. In modern practice, FAST has largely replaced DPL, but the lecture lists both — follow lecture framing for the exam.
| Concept A | Concept B | Key Discriminator |
|---|---|---|
| Neuropraxia | Axonotmesis | Neuropraxia = conduction block only, full recovery in weeks; Axonotmesis = axon dies but endoneurium intact, recovery in months |
| Chopped wound | Stab wound | Chopped = sharp edge, width ≥ depth; Stab = depth > width |
| Tension pneumothorax | Massive haemothorax | Tension = hyperresonant, tracheal deviation away; Massive = dull, can be > 1500 mL |
| Spinal shock | Neurogenic shock | Spinal shock = temporary loss of all spinal cord function below lesion (flaccid, areflexic); Neurogenic shock = cardiovascular collapse from loss of sympathetic tone (bradycardia + hypotension) |
- Open fractures (GC 231): Gustilo-Anderson classification applies when chopped wounds expose bone. Grade IIIC specifically includes vascular injury requiring repair [3].
- Hand injuries (GC 233): Detailed tendon and nerve examination of the hand; Allen's test for radial/ulnar artery patency.
- Head injury (GC 208): GCS is part of the primary survey "D" — understand scoring [4].
- Abdominal injury (GC 188): FAST scan and laparotomy thresholds for penetrating abdominal trauma [2].
- Wound healing (GC 178): Contaminated wounds from gang fights may require delayed primary closure.
- Burns lecture: May co-exist with blast injuries in the biomechanics classification.
High Yield Summary
-
ATLS ABCDE
- Always primary survey first; resuscitate simultaneously; secondary survey only after stabilisation.
-
Wound classification
- Both chopped and stabbed wounds are penetrating/sharp injuries (NOT blunt). Chopped = width ≥ depth; Stab = depth > width.
-
Documentation
- Record location, size, shape, direction, depth, wound edges, foreign bodies. Photograph/sketch before repair.
-
Partially severed artery bleeds MORE
- than completely severed (incomplete contraction/retraction → pseudoaneurysm risk).
-
Vascular repair priority
- Save life → haemostasis → proximal and distal control → primary repair or interposition vein graft → rehabilitation.
-
Seddon classification
- Neuropraxia (conduction block, full recovery), Axonotmesis (axon dies but endoneurium intact, good recovery over months), Neurotmesis (complete disruption, surgical repair needed, poor outcome).
-
Axon regrowth rate
- = 1 mm/day.
-
Chest stab wounds
- Always rule out cardiac tamponade, open pneumothorax, massive haemothorax.
-
Neck wounds deeper than platysma
- → exploration required.
-
Abdomen
- Unstable → immediate laparotomy; Stable → CT and selective management.
Active Recall - Lecture Notes
[1] Lecture slides: GC 182. Chopped and stabbed wound in gang fight Nerves and vascular injury; Classification of injuries.pdf [2] Senior notes: Maksim Surgery Notes.pdf (Section 2.1 Trauma) [3] Senior notes: Maksim Surgery Notes.pdf (Management of open fracture, Gustilo-Anderson classification) [4] Senior notes: Ryan Ho Fundamentals.pdf (Section 3.4.11 Head Injuries) [5] Senior notes: Ryan Ho Cardiology.pdf (Section 4.4.1 Acute Limb Ischaemia) [6] Senior notes: Ryan Ho Radiology.pdf (Radiology in trauma) [7] Past papers: 2020 Fourth Summative SAQ.pdf (Question 3)
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