Gram-negativeCocci

Moraxella catarrhalis

Gram-negative, oxidase-positive diplococcus that is a common cause of otitis media in children and acute exacerbations of chronic obstructive pulmonary disease in adults.

Organism Card

DomainMust know
Identity
  • G−ve diplococci; kidney-bean shaped [1]
  • Aerobic; classified with Neisseria among G−ve cocci [2][3]
  • Maltose non-fermenter (like N. gonorrhoeae; vs N. meningitidis which ferments maltose) [3]
  • Oral commensal of upper respiratory tract [1]
Lab discriminator
  • Oxidase +ve, catalase +ve
  • DNase +ve (distinguishes from Neisseria spp.)
  • Hockey-puck test: colonies can be pushed across agar plate intact
  • Maltose −ve, glucose −ve (does not ferment sugars like Neisseria)
  • Tributyrin hydrolysis +ve (vs Neisseria spp.)
Reservoir / transmission
  • Human nasopharyngeal commensal [1]
  • Colonisation rates highest in children (up to 75%)
  • Transmitted by respiratory droplets / direct contact
  • No specific Hong Kong–endemic exposure; ubiquitous worldwide
Key virulence
  • β-lactamase production → nearly universal (> 90%); penicillin/amoxicillin resistance [4]
  • Outer membrane proteins (OMPs) → adhesion to respiratory epithelium
  • LOS (lipo-oligosaccharide) → endotoxin-mediated inflammation
  • Complement resistance via surface proteins (UspA1/A2)
Clinical syndromes
  • Acute bacterial sinusitis (one of top 3 causes with S. pneumoniae, H. influenzae) [1]
  • Acute otitis media (AOM) — common paediatric pathogen [1][4]
  • Acute exacerbation of COPD (AECOPD) — important in chronic smokers [1]
  • Less common: pneumonia (elderly/immunocompromised), rarely bacteraemia
  • Triad of examinable syndromes: sinusitis + otitis media + COPD exacerbation
Diagnosis
  • Sputum Gram stain: G−ve diplococci (intracellular in PMNs)
  • Culture on blood or chocolate agar; grows at 35–37°C
  • Middle ear aspirate or sinus aspirate for definitive AOM/sinusitis diagnosis
  • Pitfall: normal flora in sputum — significance requires clinical correlation
Treatment
  • Amoxicillin-clavulanate: 1st-line for AOM / sinusitis (β-lactamase +ve strains) [4]
  • Alternatives: cefuroxime, cefpodoxime, ceftriaxone [4]
  • Macrolides (azithromycin/clarithromycin) for β-lactam allergy [4]
  • Fluoroquinolones active against M. catarrhalis [3]
  • Amoxicillin alone NOT reliable (β-lactamase producer) [4]
  • Carbapenems active but reserved for serious/resistant infections [3]
Prevention
  • No vaccine available
  • Reduce unnecessary antibiotic prescribing for URTIs (GC 106 teaching point) [5]
  • Standard droplet precautions; no special isolation or notification required
Classic traps
  • M. catarrhalis is maltose −ve like N. gonorrhoeae; N. meningitidis is maltose +ve [3]
  • Unlike Neisseria: DNase +ve, tributyrin +ve; does NOT cause STI or meningitis
  • Vignette discriminator: child with AOM or elderly COPD patient with purulent exacerbation → think M. catarrhalis when β-lactamase resistance is highlighted
  • Do NOT confuse with H. influenzae (G−ve coccobacillus, needs X+V factors)

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