Gram-negativeBacilliEnterobacterales

Yersinia enterocolitica

Gram-negative, facultatively anaerobic coccobacillus of the family Yersiniaceae that causes enterocolitis, mesenteric lymphadenitis, and pseudoappendicitis, particularly in children.

Organism Card

DomainMust know
Identity
  • G−ve coccobacillus (small rod), family Enterobacteriaceae
  • Non-lactose fermenter; facultative anaerobe
  • Motile at 25 °C (peritrichous flagella), non-motile at 37 °C (classic exam point)
  • Three pathogenic Yersinia spp: Y. pestis, Y. enterocolitica, Y. pseudotuberculosis
Lab discriminator
  • Cold enrichment: grows well at 4 °C (can isolate from refrigerated blood products) [1]
  • Urease +, oxidase −; non-lactose fermenter on MacConkey
  • CIN (cefsulodin-irgasan-novobiocin) agar → "bull's-eye" colonies
  • Motile at 22–25 °C but non-motile at 37 °C (vs other Enterobacteriaceae: motile at 37 °C)
  • Differentiate from Y. pestis: Y. enterocolitica is motile & urease + (Y. pestis is non-motile & urease −)
Reservoir / transmission
  • Reservoir: pigs (main), dogs, rodents, contaminated water
  • Transmission: fecal-oral; contaminated food (esp. undercooked pork, chitterlings)
  • Contaminated blood products (grows at 4 °C in stored RBC units → transfusion sepsis) [1]
  • No major HK-specific endemic focus but transfusion-related cases examinable
Key virulence
  • Inv (invasin): binds β1-integrin → facilitates invasion of Peyer's patches / M cells
  • Ail (attachment-invasion locus): serum resistance, epithelial invasion
  • Yersinia outer proteins (Yops) injected via Type III secretion system → antiphagocytic
  • Iron-acquisition systems (no siderophore in non-pathogenic strains); virulence enhanced by iron overload (e.g. haemochromatosis, desferrioxamine Rx)
Clinical syndromes
  • Enterocolitis: most common; watery → bloody diarrhoea, esp. young children [2]
  • Mesenteric lymphadenitis / pseudoappendicitis: RLQ pain ± fever in older children/young adults — mimics appendicitis [2][3]
  • Terminal ileitis: may mimic Crohn's disease
  • Septicaemia: esp. in iron-overload states (haemochromatosis, thalassaemia, desferrioxamine use)
  • Post-infectious sequelae: reactive arthritis (a/w HLA-B27) [4], erythema nodosum [5]
  • Classified under "penetrating" enteric infections (distal small bowel) alongside typhoidal Salmonella and C. fetus [2]
Diagnosis
  • Stool culture on CIN agar or cold-enriched culture
  • Blood culture if septicaemia suspected
  • Serology (agglutination) — limited use; cross-reacts with Brucella
  • Pitfall: routine stool culture may miss it unless lab is specifically requested (cold enrichment / CIN agar)
Treatment
  • Enterocolitis: usually self-limiting; antibiotics reserved for severe/systemic disease
  • Septicaemia / invasive: fluoroquinolones, TMP-SMX, or 3rd-gen cephalosporins
  • Key resistance: intrinsically β-lactamase producing → resistant to ampicillin and 1st-gen cephalosporins
  • Combination with aminoglycosides in severe sepsis
Prevention
  • Cook pork thoroughly; avoid raw pork products
  • Screen / leukoreduction of blood products to reduce transfusion-transmitted risk
  • No vaccine available
  • Notifiable in HK as part of bacterial foodborne illness surveillance
Classic traps
  • RLQ pain + fever in adolescent → "pseudoappendicitis" = classic vignette for Y. enterocolitica mesenteric adenitis (vs true appendicitis)
  • Transfusion reaction with septic shock from refrigerated RBC unit → think Y. enterocolitica (grows at 4 °C)
  • Iron-overload patient on desferrioxamine + sepsis → Y. enterocolitica (desferrioxamine acts as siderophore)
  • Reactive arthritis post-diarrhoea: Yersinia is one of the classic enteric triggers alongside Salmonella, Shigella, Campylobacter [4]
  • Erythema nodosum: Yersinia listed alongside Streptococcus, TB, Salmonella, Campylobacter as infective causes [5]
  • Do not confuse with Y. pestis (plague): Y. pestis is non-motile, urease −, bipolar "safety-pin" staining, transmitted by flea bites

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